Search Term: " Sodium Chloride "
3 Benefits to using Himalayan salt
Date:
April 03, 2019 10:05 AM
Many enjoy salt, but too much of it can be damaging to your health. Himalayan pink salt, on the other hand, can provide many nutritional benefits that regular table salt cannot. First, Himalayan pink salt contains more than 80 minerals to help keep you healthy, and contains overall less sodium chloride than regular table salt. Himalayan pink salt also goes through less chemical processing, which is why it is tinted pink in color. It also does not contain any food additives, unlike its table salt counterpart. Finally, many claim that Himalayan pink salt contains a saltier and bolder flavor, which is an added bonus for cooking because those who use it consume less of an amount. In sum, if you want to reduce your sodium intake, Himalayan pink salt is a great alternative. Key Takeaways:
"If you want to cut down on your sodium intake, you might want to consider switching over to Himalayan pink salt as a healthier natural salt alternative for your body’s salt needs." Read more: https://www.naturalnews.com/2019-02-05-3-benefits-to-using-himalayan-salt.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6113) Brewer's yeast, a potential probiotic?
Date:
September 07, 2018 10:52 AM
Probiotic is current trend amongst consumers and are increasingly popular for their health benefits.brewers yeast is an essential commodity for making bread and beer.A study reveals brewers yeast tolerant to various temperatures high concentration of bile salt and sodium chloride gastric juice, intestinal environment, alpha-amylase, trypsin, and lysozyme,it produces organic acids and exhibits resistance against drugs.brewers yeast can absorb cholestrol and can also produce killer toxins and also demonstrated better antibacterial activity against gram-negative bacteria than gram-positive.brewers yeast also produces enzymes that enhance nutrient utilisation in the gut. Key Takeaways:
"Ingestion of brewer’s yeast was also considered safe as it did not cause any toxic side effects based on the results of toxicity tests." Read more: https://www.naturalnews.com/2018-08-11-brewers-yeast-a-potential-probiotic.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5752) 10 Healthy alternatives to toxic processed table salt you can start using today
Date:
July 12, 2017 12:14 PM
Salt is generally not good for your body. White table salt especially is not good for you and has no health benefits. If you're going to use a salt try and go for a natural version. Pink salts or Sea salts are really great and even help keep your body's pH level good. The iodized table salt has a ton of chemicals in it and it's also bleached. The other natural salts have great benefits. Key Takeaways:
"Unlike commercial salts, naturally extracted salts like sea salts promote alkaline formation and help maintain a balanced body pH." Read more: http://www.naturalnews.com/2017-07-09-8-healthy-alternatives-to-your-toxic-table-salt-you-can-start-using-today.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4960) Difference between colloidal minerals and ionic minerals.
Date:
December 23, 2013 02:36 PM
Minerals are generally divided into two forms. Colloids Mineral Colloids are one form of minerals, where minerals are in stable form. In colloidal form, minerals are evenly distributed in the medium. Minerals in this form will remain in large and organized pattern, and thereby remain in suspension without settling down. These types of minerals are not directly absorbed by the body since they don’t have that electric charge like other minerals. The size of these minerals is also one of the reasons for this. So we can define colloids as, it is a substance, which will not diffuse easily when it is suspended in a liquid medium. Though the colloidal minerals are more dispersed in the body, the absorption is not influenced by that. In order to absorb colloidal minerals, body needs to break down these minerals into smaller units. Ionic Mineral On the other hand, ionic minerals can be easily absorbed through the human cell membranes. The main reason for this is, ionic minerals are charged and so the body needs to apply less amount of energy to get them absorbed. The colloidal minerals need to break down into smaller units to attain electric charge and thereby to get absorbed. The electric charge of ionic minerals helps them to travel from a region of higher concentration to a region of lower concentration. Atoms or group of atoms together forms the ionic minerals. They have got charge either positive or negative. During the time of absorption, the body charges the ions and makes absorption easier. Ionic minerals are more easily absorbed by the body than the colloidal minerals, since they have to go through all those process. Even after the different steps of absorption of colloidal minerals, all of them are not utilized by the body. Sodium Chloride and Potassium Chloride are 2 examples for ionic minerals. References:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=2937) Potassium And Osteoporosis - What They Have In Common!
Date:
January 19, 2011 01:51 PM
Potassium is a mineral, in the same column of the Periodic Table as sodium that it resembles. The various solutions of your body are very carefully controlled in the concentration of the electrolytes it contains, and potassium helps to maintain the electrolytic balance in your body cells. To take an example, menopausal women can tend to secrete calcium and end up suffering osteoporosis, as can older people that are not careful about their diet. That is because most people tend to use too much salt in their diet - salt is Sodium Chloride, and an excess of sodium over its ideal proportion to potassium can cause loss of certain electrolytes, of which calcium is one. Simply by increasing the amount of potassium in your diet, you can prevent or even reverse this trend. Potassium and magnesium both help calcium to be incorporated into your bone structure. Vitamin D also helps, but sodium tends to force calcium out. So by maintaining a good level of potassium in your diet you can help to prevent this condition that leads to many problems as you age.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=2221) Oxy Boost – Oxygenate your Life!
Date:
July 06, 2006 02:43 PM
Oxygen is the basis of all life and is found in the air we breathe and the water we drink. Increasing oxygen levels can have very positive effects, including improved mental clarity, endurance, immunity, better digestion and increased energy. Oxygen is the key to a healthy immune system as well as every metabolic function in our bodies. Trillion of cells use oxygen to create energy every second of every day of our lives. Atmospheric pollution and deforestation play an important role in terms of the quality of air we breathe. Oxy boost is all natural. It contains distilled water, Sodium Chloride (from sea salt), bioavailable oxygen, and essential and trace minerals. Oxy boost is the world’s premier stabilized oxygen supplement, containing one of the highest concentrations of activated oxygen available today. It is pH balanced (approximately 7.4—a perfect pH for internal and external use). This is why Oxy Boost is so unique in the field of oxygen supplements. Oxy boost has been proven in independent laboratory studies to be completely non-toxic in any concentration and at any amount. Oxy boost is safe to use both orally (sublingually or in water) and topically. Oxy boost does not depend on the digestive process to be absorbed. The body’s first line of defense A lack of sufficient oxygen in the blood stream reduces the body’s first line of defense. This has been clearly established in medical literature. Oxy boost safely increases the available oxygen levels in the bloodstream. Works with other nutrients Oxy boost can be taken with other nutritional supplements in fact; it may be safe to assume that oxy boost can even increase the efficacy of such supplements. Oxy boost works hand in hand with all other nutrients including amino acids, minerals and vitamins. These all require oxygen to be assimilated by the body and for utilization in the metabolic processes. The higher the oxygen saturation level in the blood and tissues, the more efficient and effective are the metabolic processes involving these nutrients. We recommend that Oxy boost be taken on an empty stomach, preferably 30 min before eating or two hours after eating, to get the greatest physiological benefits from the product. Oxy boost is available in regular and therapeutic (2.5 times the concentration of regular Oxy boost) strengths.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=1325) Utah's Inland Sea Minerals – Topical Application
Date:
November 22, 2005 09:23 AM
Minerals provide a bounty of healing properties that have scientifically validated their use for topical applications. These applications have been shown to have powerful local and systemic effects. The health of ones skin and hair reflects inner health. Indeed, we judge the health of animals and humans alike by their outward appearance of fur or skin, respectively. The human skin is the largest organ of the body and is highly involved in the detoxification and maintenance processes of health. Skin not only excretes and eliminates toxins; it also has a tremendous capacity to absorb health supportive substances. The pharmaceutical industry frequently takes advantage of the skin’s absorptive capacity with drug therapies. Such therapies include the transdermal delivery of drugs like nicotine, hormone patches, progesterone creams and so forth. Thus, it is apparent that natural therapies can have pronounced and powerful health effects. Clinical researchers have continued to document the clinical findings that have been observed for decades when it comes to the healing properties of topical minerals. Many of the studies on therapeutic baths have used minerals from the Dead Sea, an ancient inland sea. However, a similar and impressive array of minerals occurs in the other inland sea, the Great Salt Lake. Indeed, the high presence of magnesium from both inland seas appears to be the foremost active mineral. A comparison chart below clearly reflects the mineral analysis and similarity (see chart below). The following survey of medical research reflects a few of the many therapeutic roles for mineral salt baths. Of particular interest are the powerful effects of magnesium salts that are prevalent to both Utah’s Inland Sea and the Dead Sea that exhibit favorable effects in inflammatory disease. Arthritis: 103 patients with arthritic symptoms were treated for 1-2 weeks. They received various bath treatments with the ionic trace minerals. The study showed that the higher concentration baths offered the most impressive results. Those with the greatest physical limitation had the most pronounced improvement. Over 80 percent of the patients reported having less pain, 70 percent reported improved mobility and 60 percent were able to decrease analgesic use (i). In a different double-blind study, the use of warm mineral baths with Dead Sea salt demonstrated a lasting effect for patients suffering from degenerative arthritis. (ii) Skin: In a clinical trial conducted by a leading research university in Germany, patients with atopic (eczema) skin disorders immersed their arms in a magnesium chloride rich bath. The participants immersed one arm in tap water the other in the therapeutic magnesium rich bath. The findings showed that skin hydration was improved and skin roughness and inflammation was reduced. The researchers stated “magnesium salts are known to bind water, influence epidermal proliferation and differentiation and enhance barrier repair.” (iii) Another study showed that magnesium salts when exposed to both psoriatic and healthy skin cells provided an important anti-proliferative effect (iv). Yet another study showed that the effects of mineral baths from the Dead Sea had lasting effects for upwards of a month after treatment. (v) Head to Head Comparison (vi) (vii)
Utah’s Inland Sea Composition Dead Sea Composition By: Dr. Chris Meletis N. D.
References:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=959) Chloride: The Forgotten Essential Mineral
Date:
November 20, 2005 07:54 AM
Chloride: The Forgotten Essential Mineral Chloride is an “essential” mineral for humans. It is abundant in ionic trace mineral preparations. It is a major mineral nutrient that occurs primarily in body fluids. Chloride is a prominent negatively charged ion of the blood, where it represents 70% of the body’s total negative ion content. On average, an adult human body contains approximately 115 grams of chloride, making up about 0.15% of total body weight.1 The suggested amount of chloride intake ranges from 750 to 900 milligrams per day, based on the fact that total obligatory loss of chloride in the average person is close to 530 milligrams per day. As the principle negatively charged ion in the body, chloride serves as one of the main electrolytes of the body. Chloride, in addition to potassium and sodium, assist in the conduction of electrical impulses when dissolved in bodily water. Potassium and sodium become positive ions as they lose an electron when dissolved and chloride becomes a negative ion as it gains an electron when dissolved. A positive ion is always accompanied by a negative ion, hence the close relationship between sodium, potassium and chloride. The electrolytes are distributed throughout all body fluids including the blood, lymph, and the fluid inside and outside cells.2 The negative charge of chloride balances against the positive charges of sodium and potassium ions in order to maintain serum osmolarity. Pivotal Roles of Chloride in the Body In addition to its functions as an electrolyte, chloride combines with hydrogen in the stomach to make hydrochloric acid, a powerful digestive enzyme that is responsible for the break down of proteins, absorption of other metallic minerals, and activation of intrinsic factor, which in turn absorbs vitamin B12. Chloride is specially transported into the gastric lumen, in exchange for another negatively charged electrolyte (bicarbonate), in order to maintain electrical neutrality across the stomach membrane. After utilization in hydrochloric acid, some chloride is reabsorbed by the intestine, back into the blood stream where it is required for maintenance of extracellular fluid volume. Chloride is both actively and passively absorbed by the body, depending on the current metabolic demands. A constant exchange of chloride and bicarbonate, between red blood cells and the plasma helps to govern the pH balance and transport of carbon dioxide, a waste product of respiration, from the body. With sodium and potassium, chloride works in the nervous system to aid in the transport of electrical impulses throughout the body, as movement of negatively charged chloride into the cell propagates the nervous electrical potential. Deficiency of Chloride Deficiency of chloride is rare. However, when it does occur, it results in a life threatening condition known as alkalosis, in which the blood becomes overly alkaline. A tedious balance between alkalinity and acidity is in constant flux, and must be vigilantly maintained throughout the entire body. Alkalosis may occur as a result of excessive loss of sodium, such as heavy sweating during endurance exercise, and in cases of prolonged vomiting and diarrhea. Symptoms include muscle weakness, loss of appetite, irritability, dehydration, and profound lethargy. Hypochloremia may result from water overload, wasting conditions, and extensive bodily burns with sequestration of extracellular fluids. In a situation in which infants were inadvertently fed chloride-deficient formula, many experienced failure to thrive, anorexia, and weakness in their first year of life.3 Excess Intake? Excessive intakes of dietary chloride only occur with the ingestion of large amounts of salt and potassium chloride. The toxic effects of such diets, such as fluid retention and high blood pressure, are attributed to the high sodium and potassium levels.4 Chloride toxicity has not been observed in humans except in the special case of impaired Sodium Chloride metabolism, e.g. in congestive heart failure.5 Healthy individuals can tolerate the intake of large quantities of chloride provided that there is a concomitant intake of fresh water. Other situations in which increased blood levels of chloride are seen include diseases of improper waste elimination that occur in kidney diseases. Excess chloride is normally excreted in the urine, sweat, and bowels. In fact, excess urinary excretion of chloride occurs in high salt diets. Excessive intakes of chloride can occur in a person with compromised health in addition to an unhealthy diet. However, those that follow a healthy diet and lead an active lifestyle may need to consider supplementing their diet with this important mineral. Chloride vs. Chlorine The mineral supplement chloride is very different from the gas chlorine. While elemental chlorine is a dangerous gas that does not exist in the free elemental state in nature because of its reactivity, although it is widely distributed in combination with other elements. Chloride is related to chlorine however, as one of the most common chlorine compounds is common salt, NaCl. Chloride is a by-product of the reaction between chlorine and an electrolyte, such as potassium, magnesium, or sodium, which are essential for human metabolism. Chloride salts are essential for sustaining human metabolism and have none of the effects of isolated chlorine gas. Sources of Chloride Chloride occurs naturally in foods at levels normally less than 0.36 milligrams per gram of food. The average intake of chloride during a salt-free diet is approximately 100 milligrams per day. Unfortunately, chloride is found commonly combined with undesirable dietary sources. The most common of these negative sources is table salt. Table salt is made from a combination of sodium and chloride ions. Other unhealthful sources include yeast extracts, processed lunchmeats, and cheeses. Healthier sources of chloride include kelp (seaweed), ionic trace minerals, olives, rye, tomatoes, lettuce, and celery, although not in large enough amounts to supply the needs of an active adult.6 In its original form, however, chloride is leached from various rocks into soil and water by years of weathering processes. The chloride ion is highly mobile and is transported to closed basins, such as the Great Salt Lake, or oceans.7 Summary Chloride is a highly important, vital mineral required for both human and animal life. Without chloride, the human body would be unable to maintain fluids in blood vessels, conduct nerve transmissions, move muscles, or maintain proper kidney function. As a major electrolyte mineral of the body, chloride performs many roles, and is rapidly excreted from the body. Active adults that eat a healthy diet devoid of salt and illnesses in which vomiting and/or diarrhea are profuse warrant the supplementation of additional chloride. Replacement of chloride is essential on a daily basis to maintain regular metabolic function. Chloride is safely utilized by the body, without negative health effects. Of the negative health effects that have been associated with diets high in chloride, these are mainly attributable to the accompanying sodium and potassium, two other electrolyte minerals to which chloride is often attached -------------------------------------------------------------------------------- 1 Wesson LG. Physiology of the human kidney. New York, NY, Grune and Stratton, 1969: 591 2 Weast RC, ed. CRC handbook of chemistry and physics, 67th ed. Boca Raton, FL, CRC Press, 1986. 3 Kaleita TA. Neurologic/behavioral syndrome associated with ingestion of chloride-deficient infant formula. Pediatrics 1986 Oct;78(4):714-5 4 Beard TC. A salt-hypertension hypothesis. J Cardiovasc Pharmacol 1990;16 Suppl 7:S35-8 5 Seelig M. Cardiovascular consequences of magnesium deficiency and loss: pathogenesis, prevalence and manifestations--magnesium and chloride loss in refractory potassium repletion. Am J Cardiol 1989 Apr 18;63(14):4G-21G 6 Altschul AM, Grommet JK. Food choices for lowering sodium intake. Hypertension 1982 Sep-Oct;4(5 Pt 2):III116-20 7 Gelb SB, Anderson MP. Sources of chloride and sulfate in ground water beneath an urbanized area in Southeastern Wisconsin (Report WIS01 NTIS). Chemical abstracts, 1981, 96(2):11366g.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=958) MIGRAINE BLOCKER - Homeopathic Remedy for Headache and Migraine Relief
Date:
September 30, 2005 09:41 AM
NEW PRODUCT ANNOUNCEMENTMIGRAINE BLOCKER - Homeopathic Remedy for Headache and Migraine Relief
One tablet contains:
Suggested Use:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=802) REFERENCES
Date:
June 25, 2005 08:13 PM
REFERENCES 1 a. The Surgeon General’s “Nutrition and Health Report.” b. The Centers for Disease Control and Prevention’s “National Health and Examination Survey (NHANES III)” c. The National Academy of Science’s. Diet and Health Report: Health Promotion and Disease Objectives (DHHS Publication No. (PHS) 91-50213, Washington, DC: US Government Printing Office, 1990). e. Dietary Guidelines for Americans. 2 Rolls BJ. Carbohydrates, fats, and satiety. Am J Clin Nutr 1995; 61(4 Suppl):960S-967S. 3 McDowell MA, Briefel RR, Alaimo K, et al. Energy and macronutrient intakes of persons ages 2 months and over in the United States: Third National Health and Nutrition Examination Survey, Phase 1:1988-91. Advance data from vital and health statistics of the Centers for Disease Control and Prevention; No. 255. Hyattsville, Maryland: National Center for Health Statistics; 1994. 4 Center for Science in the Public Interest and McDonald’s Nutrition and You—A guide to Healthy Eating at McDonald’s: McDonald’s Corp,1991. 5 Bray GA. Appetite Control in Adults. In: Fernstrom JD, Miller GD eds. Appetite and Body Weight Regulation. Boca Raton: CRC Press, 1994:1-92. 6 Michnovicz JJ. How to Reduce Your Risk of Breast Cancer. New York: Warner Book Inc. 1994:54. 7 Carcinogens and Anticarcinogens in the Human Diet. National Research Council Report, National Academy of Sciences, 15 Feb. 1996. 8 Van Tallie TB. Obesity: adverse effects on health and longevity. Am J Clin Nutr 1979:32: 2723-33. 9 Somer E, M.A. R.D. Nutrition for Women. New York: Henry Hold and Company, 1993:273. 10 Swaneck GE, Fishman J. Covalent binding of the endogenous estrogen 16A-hydroxyestrone to estradiol in human breast concer cells: characterization and intranuclear localization. Proc Natl Acad Sci USA 1988:85;7831-5. 11 Colditz GA. Epidemiology of breast cancer. Findings from the nurses’ health study. Cancer1993;714:1480-9. 12 Hennen WJ. Breast Cancer Risk Reduction. The effects of supplementation with dietary indoles. Unpublished report 1992. 13 Deslypere BJ. Obesity and cancer. Metabolism 1995;44(93):24-7. 14 Somer E, M.A. R.D. Nutrition for Women. New York: Henry Hold and Company, 1993:281. 15 Whittemore AS, Kolonel LN, John M. Prostate cancer in relation to diet, physical activity, and body size in blacks, whites, and Asians in the United States and Canada. J Natl Cancer Inst 1995;87(9):629-31. 16 Key T. Risk factors for prostate cancer. Cancer Survivor 1995;23:63- 77. 17 Kondo Y, Homma Y, Aso Y, Kakizoe T. Promotional effects of twogeneration exposure to a high-fat diet on prostate carcinogenisis in ACI/Seg mice. Cancer Res 1994;54(23):6129-32. 18 Wang Y, Corr JG, Taler HT, Tao Y, Fair WR, Heston WD. Decreased growth of established human prostate LNCaP tumors in nude mice fed a low-fat diet. J Natl Cancer Inst. 1995;87(19):1456-62. 19 Nixon DW. Cancer prevention clinical trials. In-Vivo 1994;8(5):713-6. 20 Key T. Micronutrients and cancer aetiology: the epidmiological evidence. Proceed Nutr Soc 1994;53(3):605-14. 21 Gorbach SL, Goldin BR. The intestinal microflora and the colon cancer connection. Reviews of Infectious Diseases 1990;12(Suppl 2):S252-61. 22 Shrapnel WS, Calvert GD, Nestel PJ, Truswell AS. Diet and coronary heart disease. The National Heart Foundation of Australia. Med J Australia. 1995;156(Suppl):S9-S16. 23 Ellis JL, Campos-Outcalt D. Cardiovascular disease risk factors in native Americans: a literature review. Am. J. Preventive Med 1994;10(5):295-307. 24 DiBianco R. The changing syndrome of heart failure: an annotated review as we approach the 21st century. J. Hypertension 1994; 12(4 Suppl):S73- S87. 25 Van Itallie TB. Obesity: adverse effects on health and longevity. Am J Clin Nutr 1979;32(suppl):2723-33. 26 Kestin M, Moss R, Clifton PM, Nestel PJ. Comparative effects of three cereal brans on plasma lipids, blood pressure and glucose metabolism in mildly hyper-cholesterolemic men. Am J Clin Nutr 1990;52(4):661-6. 27 Story JA. Dietary fiber and lipid metabolism. In: Spiller GA, Kay RM. eds. Medical Aspects of Dietary Fiber. Penun Medical; New York, 1980, p.138. 28 Stein PP, Black HR. The role of diet in the genesis and treatment of hypertension. Med. Clin. North America. 1993;77(4):831-47. 29 Olin JW. Antihypertensive treatment in patients with peripheral vascular disease. Cleve. Clin. J. Medicine. 1994;61(5):337-44. 30 Tinker LF. Diabetes Mellitus—a priority health care issue for women. J. Am. Dietetic Association. 1994;94(9):976-85. 31 Gaspard UJ, Gottal JM, van den Brule FA. Postmenopausal changes of lipid and glucose metabolism: a review of their main aspects. Maturitas. 1995;21(3):71-8. 32 Coordt MC, Ruhe RC, McDonald RB. Aging and insulin secretion. Proc. Soc. Exp. Biology and Medicine. 1995;209(3):213-22. 33 Felber JP. From Obesity to Diabetes. Pathophysiological Considerations. Int. Journal of Obesity 1992;16:937-952. 34 Gillum RF. The association of body fat distribution with hypertension, hypertensive heart disease, coronary heart disease, diabetes, and cardiovascular risk factors in men and women age 18-79. J Chronic Diseases 1987;40:421-8. 35 Haffner SM, Stern MP, Hazuda HP, et al. Role of obesity and fat distribution in non-insulin-dependent diabetes mellits in Mexican Americans and non- Hispanic whites. Diabetes Care 1986;9:153-61. 36 Bonadonna RC, deFronzo RA. Glucose metabolism in obesity and type 2 diabetes. Diabetes and Metabolism. 1991;17(1 Pt. 2):12-35. 37 Shoemaker JK, Bonen A. Vascular actions of insulin in health and disease. Canadian J. of Applied Physiology. 1995;20(2):127-54. 38 Resnick LM. Ionic Basis of Hypertension, Insulin Resistaince, Vascular Disease, and Related Disorders. The Mechanism of ‘Syndrome X’. Am. J. Hypertension. 1993;6(suppl):123S-134S. 39 Trautwein EA. Dietetic influences on the formation and prevention of cholesterol gallstones. Z. Ernahrugswiss. 1994;33(1):2-15. 40 Cicuttini FM, Spector TD. Osteoarthritis in the aged. Epidemiological issues and optimal management. Drugs and Aging. 1995;6(5):409-20. 41 Melnyk MG, Wienstein E. Preventing obesity in black women by targeting adolescents: a literature review. J Am. Diet. Association. 1994;94(4):536-40. 42 Robinson BE, Gjerdingen Dk, Houge DR. Obesity: a move from traditional to more patient-oriented management. J. Am. Board of Family Practice. 1995;8(2):99-108. 43 Dulloo AG, Miller DS. Reversal of Obesity in the Genetically Obese fa/fa Zucker Rat with an Ehpedrine/Methylxanthines Thermogenic Mixture. J. Nutrition. 1987;117:383-9. 44 Dulloo AG, Miller DS. The thermogenic properties of ephedrin/methylxanthine mixtures: animal studies. Am J Clinical Nutr. 1986;43:388-394. 45 Richelsen B. Health risks of obesity. Significance of the regional distri-bution of adipose tissue. Ugeskr. Laeger. 1991;153(13):908-13. 46 Lissner L, Heitmann BL. Dietary fat and obesity: Evidence from epidemiology. European J. Clinical Nutrition. 1995;49(2):79-90. 47 Lissner L, Heitmann BL. The dietary fat: Carbohydrate ratio in relation to body weight, Current Opinion in Lipidology. 1995;6(1):8-13. 48 Ravussin E. Energy metabolism in obesity. Studies in the Pima Indians. Diabetes Care. 1993;16(1):232-8. 49 O’Dea K. Westernisation, insulin resistance and diabetes in Australian aborigines. Med J. Australia. 1991;155(4):258-64. 50 Bailey C. Fit or Fat . Houghton Mifflen, Boston, 1991. 51 McCarty MF. Optimizing Exercise for Fat Loss. Unpublished report. 52 Weinsier RL, Schutz Y, Bracco D. Reexamination of the relationship of resting metabolic rate and fat-free mass and the the metabolically active components of fat-free mass in humans. Am. J. Clinical Nutrition. 1992;55(4):790-4. 53 Evans WJ. Exercise, nutrition and aging. J. Nutrition. 1992;122(3 suppl):796-801. 54 Schlicker SA, Borra ST, Regan C. The weight and fitness status of United States children. Nutrition Reviews. 1994;52(1):11-7. 55 Raben A, Jensen ND, Marckmann P, Sandstrom B and Astrup A. Spontaeous weight loss during 11 weeks’ ad libitum intake of a low fat/high fiber diet in young, normal weight subjects. Stockholm Press. 1995;916-23. 56 Blundell JE, Cotton JR, Delargy H, Green S, Greenough A, King NA, Lawton, CL. The fat paradox: fat-induced satiety signals versus high fat overconsumption. Short Communication 1995:832-835. 57 Reinhold RB. Late results of gastric bypass surgery for morbid obesity. J Am Coll Nutr 1994;13(4):307-8. 58 McCredie M, Coates M Grulich A. Cancer incidence in migrants to New South Wales (Australia) from the Middle East, 1972-1991. Cancer Causes Control 1994:5(5):414-21. 59 Schiff ER, Dietschy JM. Steatorrhea Associated with Disordered Bile Acid Metabolism. Am. J. Digestive Diseases. 1969;14(6) 60 Nauss JL , Thompson JL and Nagyvary J. The binding of micellar lipids to Chitosan. Lipids. 1983;18(10):714-19. 61 Braconnot H, Sue la natrue ces champignons. Ann Chim Phys 1811;79:265. 62 Odier A. Memoire sur la composition chemique des parties cornees des insectes. Mem Soc Hist Nat Paris 1823;1:29. 63 Johnson EL, Peniston QP. Utilization of shellfish waste for chitin and Chitosan production. Chp 19 In: Chemistry and Biochemistry of Marine Food Products. Martin RE, Flick GJ, Hebard CE and Ward DR (eds.) 1982. p.415-. AVI Publishing Co., Westport, CT. 64 Shahram H. Seafood waste: the potential for industrial use. Kem Kemi 1992;19(3),256-8. 65 Rouget C. Des substances amylacees dans le tissue des animux, specialement les Articules (Chitine). Compt Rend 1859;48:792. Commission on Natural Health Products. 1995 67 Peniston QP and Johnson EL. Method for Treating an Aqueous Medium with Chitosan and Derivatives of Chitin to Remove an Impurity. US Patent 3,533,940. Oct. 30:1970. 68 Poly-D-Glucosamine (Chitosan); Exemption from the Requirement of a Tolerance. Federal Register. 1995;60(75):19523-4. Rules and Regulations. Environmental Protection Agency 40 CFR Part 180. April, 19, 1995. 69 Arul J. “Use of Chitosan films to retard post-harvest spoilage of fruits and vegetables,” Chitin Workshop. ICNHP, North Carolina State University, Raleigh, NC. 70 Karlsen J, Skaugrud O. “Excipient properties of Chitosan,” Manufacturing Chemist. 1991;62:18-9. 71 Winterowd JG, Sandford PA. Chitin and Chitosan. In: Food Polysaccharides and their Applications. Ed: Stephen AM. Marcel Dekker 1995. 72 Chitin Workshop. ICNHP, North Carolina State University, Raleigh, NC. 73 Advances in Chitin and Chitosan. Eds: CJ Brine, PA Sandford, JP Zikakis. Elsevier Applied Science. London. 1992. 74 Chitin in Nature and Technology. Eds: R Muzzarelli, C Jeuniaux, GW Gooday. Plenum Press, New York. 1986. 75 Zikakis, JP. Chitin, Chitosan and Related Enzymes. Academic Press, Inc. 1984. 76 Abelin J and Lassus A. Fat binder as a weight reducer in patients with moderate obesity. ARS Medicina, Helsinki, Aug- October, 1994. 77 Kanauchi O, Deuchi K, Imasato Y, Shizukuishi M, Kobayashi E. Increasing effect of a Chitosan and ascorbic acid mixture on fecal dietary fat excretion. Biosci Biotech Biochem 1994;58(9):1617-20. 78 Maezaki Y, Tsuji K, Nakagawa Y, et al. Hypocholesterolemic effect of Chitosan in adult males. Biosci Biotchnol Biochem1993;57(9):1439-44. 79 Kobayashi T, Otsuka S, Yugari Y. Effect of Chitosan on serum and liver cholesterol levels in cholesterol-fed rats. Nutritional Rep. Int., 1979;19(3):327-34. 80 Sugano M, Fujikawa T, Hiratsuji Y, Hasegawa Y. Hypocholesterolemic effects of Chitosan in cholesterol-fed rats. Nutr Rep. Int. 1978;18(5):531-7. 81 Vahouny G, Satchanandam S, Cassidy M, Lightfoot F, Furda I. Comparative effects of Chitosan and cholestryramine on lymphatic absorption of lipids in the rat. Am J Clin Nutr, 1983;38(2):278-84 82 Suzuki S, Suzuki M, Katayama H. Chitin and Chitosan oligomers as hypolipemics and formulations containing them. Jpn. Kokai Tokkyo Koho JP 63 41,422 [88,422] 22 Feb1988. 83 Ikeda I, Tomari Y, Sugano M. Interrelated effects of dietary fiber on lymphatic cholesterol and triglyceride absorption in rats. J Nutr 1989;119(10):1383- 7. 84 LeHoux JG and Grondin F. Some effects of Chitosan on liver function in the rat. Endocrinology. 1993;132(3):1078-84. 85 Fradet G, Brister S, Mulder D, Lough J, Averbach BL. “Evaluation of Chitosan as a New Hemostatic Agent: In Vitro and In Vivo Experiments In Chitin in Nature and Technology. Eds: R Muzzarelli, C Jeuniaux, GW Gooday. Plenum Press, New York. 1986. 86 Malette W, Quigley H, Gaines R, Johnson N, Rainer WG. Chitosan A New Hemostatic. Annals of Thorasic Surgery. 1983;36:55. 87 Malette W, Quigley H, Adickes ED. Chitosan effect in Vascular Surgery, Tissue Culture and Tissue Regeneration. In R Muzzarelli, C Jeuniaux, GW Gooday, Eds: Chitin in Nature and Technology. Plenum Press, New York. 1986. 88 Okamoto Y, Tomita T, Minami S, et al. Effects of Chitosan on experimental abscess with Staphylococcus aureus in dogs. J. Vet. Med., 1995;57(4):765-7. 89 Klokkevold PR, Lew DS, Ellis DG, Bertolami CN. Effect of Chitosan on lingual hemostasis in rabbits. Journal of Oral-Maxillofac-Surg, 1991;Aug. 49(8):858-63. 89 Surgery, Tissue Culture and Tissue Regeneration. In Chitin in Nature and Technology. Eds: R Muzzarelli, C Jeuniaux, GW Gooday. Plenum Press, New York. 1986. 90 Hiroshi S, Makoto K, Shoji A, Yoshikazu S. Antibacterial fiber blended with Chitosan. Sixth International Conference on Chitin and Chitosan. Sea Fisheries Institute, Gdynia, Poland. August 1994;16-19. 91 Shimai Y, Tsukuda K, Seino H. Antiacne preparations containing chitin, Chitosan or their partial degradation products. Jpn. Kikai Tokkyo Koho JP 04,288,017 [92,288,017] 13 Oct 1992. 92 Suzuki K, Okawa Y, Suzuki S, Suzuki M. Candidacidal effect of peritoneal exudate cells in mice administered with chitin or Chitosan: the role of serine protease in the mechanism of oxygen-independent candidacidal effect. Microbiol Immunol. 1987;31(4):375-9. 93 Sawada G, Akaha Y, Naito H, Fujita M. Synergistic food preservatives containing organic acids, Chitosan and citrus seed extracts. Jpn, Kokai Kokkyo Koho JP 04 27,373 [92 27,373] 30 Jan 1992. 94 Min H-K, Hatai K, Bai S. Some inhibitory effects of Chitosan on fishpathogenic oomycete, Saprolegnia parasitic. Gyobyo Kenkyu, 1994;29(2):73-4. 95 Nelson JL, Alexander JW, Gianotti L, Chalk CL, Pyles T. The influence of dietary fiber on microbial growth in vitro and bacterial translocation after burn injury in mice. Nutr 1994;10(1):32-6. 96 Ochiai Y, Kanazawa Y. Chitosan as virucide. Jpn Kokai Tokkyo Koho 79 41,326. 97 Hillyard IW, Doczi J, Kiernan. Antacid and antiulcer properties of the polysaccharide Chitosan in the rat. Proc Soc Expl Biol Med 1964; 115:1108-1112. 98 Shibasaki K, Sano H, MatsukuboT, Takaesu Y. pH response of human dental plaque to chewing gum supplemented with low molecular Chitosan. Bull- Tokyo-Dent-Coll, 1994:35(2): 61-6. 99 Kato H, Okuda H. Chitosan as antihypertensive. Jpn. Kikoi Tokyo Koho JP 06 56,674 [94 56,674] 100 Kato H, Taguchi T. Mechanism of the rise in blood pressure by Sodium Chloride and decrease effect of Chitosan on blood pressure. Baiosaiensu to Indasutori 1993;51(12):987-8. 101 Muzzarelli R, Biagini G, Pugnaoni A, Filippini O, Baldassarre V, Castaldini C, and Rizzoli C. Reconstruction of Periodontal Tissue with Chitosan. Biomaterials. 1989;10:598-603. 102 Sapelli P, Baldassarre V, Muzzarelli R, Emanuelli M. Chitosan in Dentistry. In Chitin in Nature and Technology. Eds: R Muzzarelli, C Jeuniaux, GW Gooday. Plenum Press, New York. 1986. 103 Borah G, Scott G, Wortham K. Bone induction by Chitosan in endochrondral bones of the extremities. In Advances in Chitin and Chitosan. Eds: CJ Brine, PA Sandford, JP Zikakis. Elsevier Applied Science. London. 1992. 104 Ito F. Role of Chitosan as a supplementary food for osteoporosis. Gekkan Fudo Kemikaru, 1995;11(2):39-44. 105 Nakamura S, Yoshioka T, hamada S, Kimura I. Chitosan for enhancement of bioavailability of calcium. Jpn. Kokai Tokkyo Koho JP 07 194,316 [95 194,316] 01 Aug 1995. 106 Maekawa A, Wada M. Food Containing chitin or its derivatives for reduction of blood and urine uric acid. Jpn. Kokai Tokkyo Koho JP 03 280,852 [91 280,852], 11 Dec 1991. 107 Weisberg M, Gubner R. Compositions for oral administration comprising Chitosan and a pharmaceutically acceptable carrier. Antacid preparations for alleviating gastric hyperacidity. U.S. patent 3257275 108 Kanauchi O, Deuchi K, Imasato Y, Shizukuishi M, Kobayashi E. Mechanism for the inhibition of fat digestion by Chitosan and for the synergistic effect of ascorbate. Biosci Biotech Biochem1995;59(5):786-90. 109 McCausland CW. Fat Binding Properties of Chitosan as Compared to Other Dietary Fibers. Private communication. 24 Jan1995. 110 Deuchi K, Kanauchi O, Imasato Y, Kobayashi E. Biosci Biotech Biochem. 1994:58,1613-6. 111 Ebihara K, Schneeman BO. Interaction of bile acids, phospholipids, cholesterol and triglyceride with dietary fibers in the small intestine of rats. J Nutr 1989;119(8):1100-6. 112 Weil A, M.D. Natural Health Natural Medicine: Boston: Houghton Mifflin, 1990:182. 113 Chen Y-H, Riby Y, Srivastava P, Bartholomew J, Denison M, Bjeldanes L. Regualtion of CYP1A1 by indolo[3,2-b]carbazole in murine hepatoma cells. J Biol Chem 1995;270(38):22548-55. 114 Intestinal Absorption of metal ions and chelates. Ashmead HD, Graff DJ, Ashmead HH. Charles C Thomas, Springfield, IL 1985. 115 Nutrient Interactions. Bodwell CE, Erdman JW Jr. Marcel Dekker New York 1988. 116 Heleniak EP, Aston B. Prostaglandins, Brown Fat and Weight Loss. Medical Hypotheses 1989;28:13-33. 117 Connor WE, DeFrancesco CA, Connor SL. N-3 fatty acids from fish oil. Effects on plasma lipoproteins and hypertriglyceridemic patients. Ann NY Acad Sci 1993;683:16-34. 118 Conte AA. A non-prescription alternative in weight reduction therapy. The Bariatrician Summer 1993:17-19. 119 McCarty MF. Inhibition of citrate lyase may aid aerobic endurance. Unpublished manuscript. 120 Bray GA. Weight homeostasis. Annual Rev Med 1991;42:205-216. 121 Dulloo AG, Miller DS. The thermogenic properties of Ephedrin/Methylxanthine mixtures: Human studies. Intl J Obesity 986;10:467-481. 122 Arai K, Kinumaki T, Fujita, T. Bulletin Tokai Regional Fisheries Res Lab. 1968;No. 56. 123 Bough WA. Private communication. 124 Freidrich EJ, Gehan, EA, Rall DP, Schmidt LH, Skipper HE. Cancer Chemotherapy Reports 1966;50(4):219-244. 125 A Drovanti, AA Bignamini, AL Rovati. Therapeutic activity of oral glucosamine sulfate in osteoarthritis: A placebo-controlled double-blind investigation. Clinical Therapeutics 1980;3(4):260-272. 126 K Deuchi, O Kanauchi, M Shizukuishi, E Kobayashi. Continuous and massive intake of Chitosan affects mineral and fat-soluble vitamin status in rats fed on a high-fat diet. Biosci. Biotech. Biochemistry. 1995;59(7):1211-6. 127 . BesChitin W in Chitin Wound Healing (video), Unitika Corporation, April 1992.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=507) The Science of Healthy Hair
Date:
June 10, 2005 03:44 PM
The Science of Healthy Hair by Susan Weiner Energy Times, January 5, 2002 From the strength-giving mane of Sampson to the magically long locks of Rapunzel, hair has had the power to captivate since biblical times. Today, its lure is just as compelling and hair remains an important form of self-expression and self-image. A healthy head of hair is more than an asset to your appearance. A hairstyle can reflect a mood, an attitude or a personal style, while unkempt hair may reveal the status of one's emotional or physical health. Even a "good" hair day vs. a "bad" hair day can significantly determine how your frame of mind takes shape. We can't always control the frizz factor or the humid weather that makes our curls fall flat, but many natural approaches are available to allow us to put our best looking follicle forward. Whether your hair is sleek and stylish, long and slinky, spiky punk rock-hip or wash-and-wear, botanical-based products and proper nutrition can bring out the very best in your locks. Don't Fool Mother Nature No matter how often you cut, dye, perm or blow-dry your hair, Mother Nature, with the help of your DNA, has blessed you with a quite specific quality and quantity of hair. Styling may work to change the appearance of your hair, but nothing can change your genetics. Every hair on your body, from the soft down on your arms to the coarser, longer hairs on your head, grows from a cell-lined indentation called a follicle. The hair follicle consists of three cylinders; the central cylinder determines whether your hair is straight, wavy or curly. Each hair shaft alternately grows or goes into a dormant phase. "At any one time, approximately fifteen percent of the one hundred thousand or so hairs on the head are resting, while the rest are growing or lengthening," say Arthur Balin, MD, PhD, and Loretta Pratt Balin, MD (The Life of the Skin: What It Hides, What It Reveals, and How It Communicates, Bantam). Hair constantly comes and goes, falling out consistently even when it is healthy. Consequently, a normal head can shed up to one hundred resting-phase hairs a day. When hair is subjected to harsh chemicals and treatment, even more may fall out. If you're concerned with hair loss, gently pull on a small section of hair; if fewer than five hairs come out, hair loss is within normal range. What's Your Type? Normal hair is an elusive commodity in these stressed-out days of over-washed, over-dried and chemically treated hair. If your tresses look frizzy, tangle easily or generally lack moisture, they're probably dry. Dry hair lacks the proper oil content to maintain an ample sheen and is usually dull-looking. To gain back a natural shine, cut back on shampooing and use a natural conditioner formulated for dry hair. Look for essential oils such as jojoba, evening primrose, blue chamomile, and white camellia, and B vitamins (such as panthenol) and aloe vera, suggests Aubrey Hampton, founder of Aubrey Organics. Drinking plenty of water, eating a diet that's not ultra-low in fats and using a humidifier may also help improve dull-looking dry hair, points out David E. Bank, MD (Beautiful Skin: Every Woman's Guide to Looking Her Best at Any Age, Adams Media). (Excessively dry hair may be a significant sign of metabolic disease. If you don't notice a marked improvement in your scalp after taking measures to improve dry hair, or your hair is abnormally dry, consult your health practitioner to see if stronger cures should be implemented.) Too Much Oil Hair that appears greasy within 24 hours after shampooing is oily. In that case, try gentle shampoos and herbal rinses with essential oils including quillaya bark, amino acids mixed with saponins, non-coloring henna and peppermint. For an oily scalp and dry ends, condition only the ends. Styling products should be oil-free. For thin or flyaway hair, products with natural thickening agents such as panthenol can help pump up the volume. Color treated and damaged hair can benefit from sulfur-containing amino acids; check your natural foods store for hair care products that contain horsetail, coltsfoot and cysteine. Tea tree oil products are effective when you are trying to control dandruff and a problem scalp. The Must-To-Avoids If the label lists sodium lauryl sulfate, steer clear, warns Hampton. And, says Dr. Bank, sodium C-14-16 olefin sulfonate, a harsh chemical found in cheap shampoos, is the worst of the worst when it comes to offensive hair care ingredients. "You also need to watch out for Sodium Chloride-table salt-in the ingredient list. It's a cheap ingredient to thicken shampoo and strips the hair of oils." Feed Your Head To optimize shine and fullness, improve your nutrition, says Bruce Miller, MD, author of The Nutrition Guarantee (Summit Publishing Group). "Good nutrition is as essential to healthy, attractive hair as it is to clear, glowing skin," notes Dr. Miller. "Your hair directly reflects your care and feeding of it." Your hair consists of about 97% protein, containing nineteen of the twenty-two amino acids that form protein, explains Dr. Miller. If you skimp on quality protein, your hair may reflect this amino acid imbalance by breaking, cracking and splitting. Hair follicles pass on the nutrients you consume, nourishing the new cells that form the growing hair shaft. As the hair gradually pushes upward, the shaft is continually lubricated by the busy sebaceous glands. For a smoother transition through the shaft and undamaged hair, lecithin provides a welcome dose of lubrication, as well as the important B vitamins choline and inositol, vital to healthy hair. In fact, the B vitamins are crucial to the growth of full bodied, healthy hair. The B complex strengthens, forms and smoothes the hair shafts, and helps maintain an even hair color, even warding off the beginning of gray hair. For thick and shiny hair, vitamin A works in conjunction with the B vitamins. Zinc can strengthen the hair shafts by thickening them. Thicker and stronger hair shafts increase your chances of holding on to your hair and suffering fewer lost hairs. When it comes to hair retention, genetics count. The more hair your parents retained, the greater your chance of keeping yours. Think Diet If you're interested in optimal hair health, think nutrition. Eating for the sake of your curls is a lot like eating for overall health: plenty of fresh fruits and vegetables, healthy grains and lean sources of protein, including tofu and other soy-based foodstuffs. To support healthy hair, some experts advocate foods high in biotin, including brown rice, brewer's yeast, bulgur, green peas, lentils, oats, soybeans, sunflower seeds and walnuts. The natural phytochemicals in green tea may aid hair, while ginkgo biloba improves circulation to the scalp. Don't forget your daily vitamins and be sure to take an iron and B12 supplement. Chinese Treatment Herbs from China show great promise for helping hair. He Shou Wu, made from Polygoni multiflori (the eastern wild rose), is reputed by devotees to restore color, slow hair loss, and help hair grow back. In Chinese medicine, this botanical has been used as an adaptogen to boost overall health and longevity. Within the context of Traditional Chinese Medicine (TCM), He Shou Wu is supposed to strengthen the liver and kidney meridians and support healthy blood. Many Asians use the herb to promote higher levels of qi, the TCM concept that encompasses your life's overall energy. Show a Little Tenderness Long-term exposure to sunlight and seawater can damage hair, as can combing or brushing wet hair. Treat your hair with kid gloves, use natural products that are gentle on hair, and avoid chemical treatments. If you're looking to lose weight, avoid crash diets; a sudden drop in nutrition can cause deficiencies and lead to hair damage and loss. Keeping a wonderful head of hair means staying ahead of the curve with proper nutrition, the right supplements and a continuous program of TLC. In that way, you can maintain the crowning head of hair you've always coveted.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=271) |